AP39 and AP123 promoted Nrf2-regulated redox signaling pathway

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ap123 me AP39 and AP123 protect against UVA-induced apoptotic cells and oxidative stress NHDFs were treated with AP39 and AP123 or nontargeted controls, ADT-OH and AP123 treatment rescued eNOS expression, as well as NO levels, and restored p-CREB expression in both the HG environment and the presence of PAG Highlights • Mitochondrial targeted H2S donor AP123 restores endothelial function through a mechanism involving PI3KCREBeNOS axis • H2S availability is

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